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Academic Journal

Cathepsin S is not crucial to TSHR processing and presentation in a murine model of Graves' disease.

Subjects: *Peptides; *Proteolytic enzymes; *Autoimmune diseases

  • Source: Immunology. Dec2005, Vol. 116 Issue 4, p532-540. 9p. 4 Graphs.

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Academic Journal

Cathepsin S is not crucial to TSHR processing and presentation in a murine model of Graves' disease

Subjects: 0301 basic medicine; Antigen Presentation; Mice, Inbred BALB C

  • Source: Immunology. 116:532-540

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Academic Journal

Clonal relationships between thyroid-stimulating hormone receptor-stimulating antibodies illustrate the effect of hypermutation on antibody function.

  • Authors : Padoa CJ; Division of Gene and Cell Based Therapy, King's College London School of Medicine, London, UK.; Larsen SL

Subjects: Antibodies, Monoclonal*; Graves Disease/Graves Disease/Graves Disease/*immunology ; Immunoglobulin Heavy Chains/Immunoglobulin Heavy Chains/Immunoglobulin Heavy Chains/*genetics

  • Source: Immunology [Immunology] 2010 Feb; Vol. 129 (2), pp. 300-8. Date of Electronic Publication: 2009 Oct 21.Publisher: Blackwell Scientific Publications Country of Publication: England NLM ID: 0374672 Publication Model: Print-Electronic Cited Medium: Internet

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Academic Journal

Anti‐Gal: an abundant human natural antibody of multiple pathogeneses and clinical benefits

Subjects: 0301 basic medicine; Transplantation, Heterologous; Medical Immunology

  • Source: Immunology. 140:1-11

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Academic Journal

Soluble L‐selectin levels in type I diabetes mellitus: a surrogate marker fordisease activity?

Subjects: Adult; CD4-Positive T-Lymphocytes; Aging

  • Source: Immunology. 99:320-325

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Academic Journal

Chronic exposure in vivo to thyrotropin receptor stimulating monoclonal antibodies sustains high thyroxine levels and thyroid hyperplasia in thyroid autoimmunity‐prone HLA‐DRB1*0301 transgenic mice

Subjects: Male; Autoimmunity; Mice, Transgenic

  • Source: Immunology. 122:261-267

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Academic Journal

Hereditary deficiency of OKT4-positive cells: studies for mode of inheritance and lymphocyte functions.

Subjects: Antibodies, Monoclonal/Antibodies, Monoclonal/Antibodies, Monoclonal/*immunology ; Graves Disease/Graves Disease/Graves Disease/*immunology ; Immunologic Deficiency Syndromes/Immunologic Deficiency Syndromes/Immunologic Deficiency Syndromes/*genetics

  • Source: Immunology [Immunology] 1984 Dec; Vol. 53 (4), pp. 643-9.Publisher: Blackwell Scientific Publications Country of Publication: England NLM ID: 0374672 Publication Model: Print Cited Medium: Print ISSN:

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Academic Journal

Expression and function of multiple regulators of complement activation in autoimmune thyroid disease

Subjects: Complement Inactivator Proteins; Membrane Glycoproteins; CD55 Antigens

  • Source: Immunology. 81(4)

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Academic Journal

Homotherapy for heteropathy: Interleukin‐41 and its biological functions.

Subjects: *Immunologic diseases; *Crohn's disease; *Autoimmune hepatitis

  • Source: Immunology. Sep2024, Vol. 173 Issue 1, p1-13. 13p.

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Academic Journal

Inhibitory effects of nicotinamide on intercellular adhesion molecule-1 expression on cultured human thyroid cells

Subjects: 0301 basic medicine; Niacinamide; Dose-Response Relationship, Immunologic

  • Source: Immunology. 80(2)

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  • 1-10 of  69 results for ""Graves Disease""