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Academic Journal

Conditional Deletion of Sost in MSC-Derived Lineages Identifies Specific Cell-Type Contributions to Bone Mass and B-Cell Development.

Authors : Yee, Cristal S; Manilay, Jennifer O; Chang, Jiun C

Subjects: Femur; Lumbar Vertebrae; B-Lymphocytes

Source: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 33(10)

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Academic Journal

Contributions of Material Properties and Structure to Increased Bone Fragility for a Given Bone Mass in the UCD-T2DM Rat Model of Type 2 Diabetes.

Authors : Acevedo, Claire; Sylvia, Meghan; Schaible, Eric

Subjects: Bone and Bones; Animals; Rats

Source: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 33(6)

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Academic Journal

Blockade of receptor-activated G(i) signaling in osteoblasts in vivo leads to site-specific increases in cortical and cancellous bone formation.

Authors : Millard, Susan M; Louie, Alyssa M; Wattanachanya, Lalita

Subjects: Femur; Osteoclasts; Osteoblasts

Source: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 26(4)

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Academic Journal

Differential maintenance of cortical and cancellous bone strength following discontinuation of bone-active agents.

Authors : Shahnazari, Mohammad; Yao, Wei; Wang, Bob

Subjects: Bone and Bones; Femur; Tibia

Source: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 26(3)

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Prolonged treatments with antiresorptive agents and PTH have different effects on bone strength and the degree of mineralization in old estrogen-deficient osteoporotic rats.

Authors : Cheng, Zhiqiang; Yao, Wei; Zimmermann, Elizabeth A

Subjects: Bone and Bones; Tibia; Lumbar Vertebrae

Source: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 24(2)

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Conditional Deletion of Sost in MSC-Derived Lineages Identifies Specific Cell-Type Contributions to Bone Mass and B-Cell Development.

Contributions of Material Properties and Structure to Increased Bone Fragility for a Given Bone Mass in the UCD-T2DM Rat Model of Type 2 Diabetes.

Blockade of receptor-activated G(i) signaling in osteoblasts in vivo leads to site-specific increases in cortical and cancellous bone formation.

Differential maintenance of cortical and cancellous bone strength following discontinuation of bone-active agents.

Prolonged treatments with antiresorptive agents and PTH have different effects on bone strength and the degree of mineralization in old estrogen-deficient osteoporotic rats.

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