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Academic Journal

GRPR-induced FAM135A expression promote perineural invasion in prostate cancer.

  • Authors : Zhang N; Department of Urology, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Ruan X

Subjects: Prostatic Neoplasms*/Prostatic Neoplasms*/Prostatic Neoplasms*/pathology ; Prostatic Neoplasms*/Prostatic Neoplasms*/Prostatic Neoplasms*/metabolism ; Prostatic Neoplasms*/Prostatic Neoplasms*/Prostatic Neoplasms*/genetics

  • Source: Molecular cancer [Mol Cancer] 2025 Nov 17; Vol. 24 (1), pp. 293. Date of Electronic Publication: 2025 Nov 17.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

Epigenetic modification of cuproptosis by non-coding RNAs in cancer drug resistance.

  • Authors : Du F; Department of Pharmacy, The Fourth Affiliated Hospital Of Southwest Medical University, 620000, Meishan, Sichuan, PR China.; Tang L

Subjects: Drug Resistance, Neoplasm*/Drug Resistance, Neoplasm*/Drug Resistance, Neoplasm*/genetics ; Epigenesis, Genetic* ; RNA, Untranslated*/RNA, Untranslated*/RNA, Untranslated*/genetics

  • Source: Molecular cancer [Mol Cancer] 2025 Sep 25; Vol. 24 (1), pp. 230. Date of Electronic Publication: 2025 Sep 25.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

CircZFR/YTHDF3 axis drives lymph node metastasis in cervical cancer via FASN translation.

  • Authors : Zhou M; Department of Gynecology, Cancer Hospital of China Medical University, Cancer Hospital of Dalian University of Technology, Liaoning Cancer Hospital & Institute, No.44 Xiaoheyan Road, Dadong District, Shenyang, 110042, Liaoning Province, China.; Gao Y

Subjects: Uterine Cervical Neoplasms*/Uterine Cervical Neoplasms*/Uterine Cervical Neoplasms*/genetics ; Uterine Cervical Neoplasms*/Uterine Cervical Neoplasms*/Uterine Cervical Neoplasms*/pathology ; Uterine Cervical Neoplasms*/Uterine Cervical Neoplasms*/Uterine Cervical Neoplasms*/metabolism

  • Source: Molecular cancer [Mol Cancer] 2025 Aug 21; Vol. 24 (1), pp. 218. Date of Electronic Publication: 2025 Aug 21.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

STK3 is a transcriptional target of YAP1 and a hub component in the crosstalk between Hippo and Wnt signaling pathways during gastric carcinogenesis.

  • Authors : Xie F; Department of Anatomical and Cellular Pathology, State Key Laboratory of Translational Oncology, Prince of Wales Hospital, Sir Y.K. Pao Cancer Center, The Chinese University of Hong Kong, Hong Kong, China.; Institute of Digestive Disease, State Key Laboratory of Digestive Disease, Li Ka Shing Institute of Health Science, The Chinese University of Hong Kong, Hong Kong, China.

Subjects: Stomach Neoplasms*/Stomach Neoplasms*/Stomach Neoplasms*/metabolism ; Stomach Neoplasms*/Stomach Neoplasms*/Stomach Neoplasms*/pathology ; Stomach Neoplasms*/Stomach Neoplasms*/Stomach Neoplasms*/genetics

  • Source: Molecular cancer [Mol Cancer] 2025 Jul 02; Vol. 24 (1), pp. 186. Date of Electronic Publication: 2025 Jul 02.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

CREPT is required for the metastasis of triple-negative breast cancer through a co-operational-chromatin loop-based gene regulation.

  • Authors : Li J; State Key Laboratory of Membrane Biology, School of Basic Medical Sciences, School of Medicine, Tsinghua University, Beijing, 100084, China.; Xu L

Subjects: Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/genetics ; Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/pathology ; Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/metabolism

  • Source: Molecular cancer [Mol Cancer] 2025 Jun 10; Vol. 24 (1), pp. 170. Date of Electronic Publication: 2025 Jun 10.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

Profiling triple-negative breast cancer-specific super-enhancers identifies high-risk mesenchymal development subtype and BETi-Targetable vulnerabilities.

  • Authors : Chen QS; State Key Laboratory of Oncology in South China, Guangdong Provincial Clinical Research Center for Cancer, Sun Yat-sen University Cancer Center, Guangzhou, 510060, P. R. China.; Department of Breast Oncology, Sun Yat-sen University Cancer Center, Guangzhou, China.

Subjects: Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/genetics ; Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/pathology ; Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/Triple Negative Breast Neoplasms*/drug therapy

  • Source: Molecular cancer [Mol Cancer] 2025 May 13; Vol. 24 (1), pp. 141. Date of Electronic Publication: 2025 May 13.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

Circular RNA circDCUN1D4 suppresses hepatocellular carcinoma development via targeting the miR-590-5p/ TIMP3 axis.

  • Authors : Li H; College of Chemistry and Lie Science, Beijing University of Technology, Beijing, 100124, China. .; Allife Medical Science and Technology Co., Ltd. Economic and Technological Development Zone, Beijing, 100176, China. .

Subjects: MicroRNAs*/MicroRNAs*/MicroRNAs*/genetics ; RNA, Circular*/RNA, Circular*/RNA, Circular*/genetics ; Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/genetics

  • Source: Molecular cancer [Mol Cancer] 2025 Mar 25; Vol. 24 (1), pp. 95. Date of Electronic Publication: 2025 Mar 25.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

Lactate accumulation induces H4K12la to activate super-enhancer-driven RAD23A expression and promote niraparib resistance in ovarian cancer.

  • Authors : Lu B; Department of Obstetrics and Gynecology, Department of Gynecologic Oncology Research Office, Guangzhou Key Laboratory of Targeted Therapy for Gynecologic Oncology, Guangdong Provincial Key Laboratory of Major Obstetric Diseases, Guangdong Provincial Clinical Research Center for Obstetrics and Gynecology, Guangdong-Hong Kong-Macao Greater Bay Area Higher Education Joint Laboratory of Maternal-Fetal Medicine, The Third Affiliated Hospital, Guangzhou Medical University, No.63 Duobao Raod, Liwan District, Guangzhou, Guangdong Province, P. R. China.; Chen S

Subjects: Ovarian Neoplasms*/Ovarian Neoplasms*/Ovarian Neoplasms*/metabolism ; Ovarian Neoplasms*/Ovarian Neoplasms*/Ovarian Neoplasms*/drug therapy ; Ovarian Neoplasms*/Ovarian Neoplasms*/Ovarian Neoplasms*/genetics

  • Source: Molecular cancer [Mol Cancer] 2025 Mar 19; Vol. 24 (1), pp. 83. Date of Electronic Publication: 2025 Mar 19.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

Quantitative proteomic analysis unveils a critical role of VARS1 in hepatocellular carcinoma aggressiveness through the modulation of MAGI1 expression.

  • Authors : Hermán-Sánchez N; Department of Cell Biology, Physiology, and Immunology, University of Córdoba, CIBER Pathophysiology of Obesity and Nutrition (CIBERobn), Córdoba, 14004, Spain.; Maimónides Institute of Biomedical Research of Córdoba (IMIBIC), Reina Sofía University Hospital, Córdoba, 14004, Spain.

Subjects: Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/pathology ; Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/metabolism ; Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/genetics

  • Source: Molecular cancer [Mol Cancer] 2025 Jan 14; Vol. 24 (1), pp. 15. Date of Electronic Publication: 2025 Jan 14.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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Academic Journal

Mir-483-5p-mediated activating of IGF2/H19 enhancer up-regulates IGF2/H19 expression via chromatin loops to promote the malignant progression of hepatocellular carcinoma.

  • Authors : Chen W; Department of Gastroenterology, The First Affiliated Hospital, Jinan University, Guangzhou, Guangdong, 510630, P. R. China.; Department of Gastroenterology, The First People's Hospital of Zunyi, (The Third Affiliated Hospital of Zunyi Medical University), Zunyi, P. R. China.

Subjects: MicroRNAs*/MicroRNAs*/MicroRNAs*/genetics ; Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/genetics ; Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/Carcinoma, Hepatocellular*/pathology

  • Source: Molecular cancer [Mol Cancer] 2025 Jan 11; Vol. 24 (1), pp. 10. Date of Electronic Publication: 2025 Jan 11.Publisher: BioMed Central Country of Publication: England NLM ID: 101147698 Publication Model: Electronic Cited Medium: Internet ISSN: 1476-4598

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  • 1-10 of  2,787 results for ""Gene Expression Regulation""