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Academic Journal

MTHFD2 promotes osteoclastogenesis and bone loss in rheumatoid arthritis by enhancing CKMT1-mediated oxidative phosphorylation.

Authors : Li, Yujing¹ (AUTHOR); Cai, Minglong¹ (AUTHOR); Qin, Yi^1,2 (AUTHOR)

Source: BMC Medicine. 2/27/2025, Vol. 23 Issue 1, p1-17. 17p.

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Up-regulation of MTHFD2 is associated with clinicopathological characteristics and poor survival in ovarian cancer, possibly by regulating MOB1A signaling.

Authors : Cui, Xiangrong^1,2 (AUTHOR); Su, Huancheng² (AUTHOR); Yang, Jiaolin² (AUTHOR)

Source: Journal of Ovarian Research. 2/8/2022, Vol. 15 Issue 1, p1-14. 14p.

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PPFIA4 promotes castration-resistant prostate cancer by enhancing mitochondrial metabolism through MTHFD2.

Authors : Zhao, Ru¹ (AUTHOR); Feng, Tingting¹ (AUTHOR); Gao, Lin¹ (AUTHOR)

Source: Journal of Experimental & Clinical Cancer Research (17569966). 4/5/2022, Vol. 41 Issue 1, p1-17. 17p.

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MTHFD2 is a potential oncogene for its strong association with poor prognosis and high level of immune infiltrates in urothelial carcinomas of bladder.

Authors : Zhu, Lin¹ (AUTHOR); Liu, Xianhui¹ (AUTHOR); Zhang, Weiyu^1,2 (AUTHOR)

Source: BMC Cancer. 5/17/2022, Vol. 22 Issue 1, p1-16. 16p.

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Metabolic subtypes and immune landscapes in esophageal squamous cell carcinoma: prognostic implications and potential for personalized therapies.

Authors : Yu, Xiao-wan¹ (AUTHOR) ; She, Pei-wei^2,3 (AUTHOR); Chen, Fang-chuan⁴ (AUTHOR)

Source: BMC Cancer. 2/19/2024, Vol. 24 Issue 1, p1-16. 16p.

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Mitochondrial folate pathway regulates myofibroblast differentiation and silica-induced pulmonary fibrosis.

Authors : Qu, Yaqian^1,2 (AUTHOR); Zhai, Ruonan^1,3,4 (AUTHOR); Wang, Dandan^3,4 (AUTHOR)

Source: Journal of Translational Medicine. 6/6/2023, Vol. 21 Issue 1, p1-12. 12p.

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Pharmacological targeting of one‑carbon metabolism as a novel therapeutic strategy for glioblastoma.

Authors : Sun, Yanfei¹ (AUTHOR); Mu, Guangjing¹ (AUTHOR); Xue, Zhiwei¹ (AUTHOR)

Source: Journal of Translational Medicine. 6/29/2023, Vol. 21 Issue 1, p1-4. 4p.

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Glioma cells require one-carbon metabolism to survive glutamine starvation.

Authors : Tanaka, Kazuhiro¹ (AUTHOR) ; Sasayama, Takashi¹ (AUTHOR); Nagashima, Hiroaki¹ (AUTHOR)

Source: Acta Neuropathologica Communications. 1/19/2021, Vol. 9 Issue 1, p1-14. 14p.

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MYEOV overexpression induced by demethylation of its promoter contributes to pancreatic cancer progression via activation of the folate cycle/c-Myc/mTORC1 pathway.

Authors : Tange, Shoichiro¹ (AUTHOR); Hirano, Tomomi¹ (AUTHOR); Idogawa, Masashi^1,2 (AUTHOR)

Source: BMC Cancer. 1/25/2023, Vol. 23 Issue 1, p1-12. 12p.

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Comparative profiling of single-cell transcriptome reveals heterogeneity of tumor microenvironment between solid and acinar lung adenocarcinoma.

Authors : Li, Dianke¹ (AUTHOR); Yu, Huansha² (AUTHOR); Hu, Junjie¹ (AUTHOR)

Source: Journal of Translational Medicine. 9/23/2022, Vol. 20 Issue 1, p1-21. 21p.

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MTHFD2 promotes osteoclastogenesis and bone loss in rheumatoid arthritis by enhancing CKMT1-mediated oxidative phosphorylation.

Up-regulation of MTHFD2 is associated with clinicopathological characteristics and poor survival in ovarian cancer, possibly by regulating MOB1A signaling.

PPFIA4 promotes castration-resistant prostate cancer by enhancing mitochondrial metabolism through MTHFD2.

MTHFD2 is a potential oncogene for its strong association with poor prognosis and high level of immune infiltrates in urothelial carcinomas of bladder.

Metabolic subtypes and immune landscapes in esophageal squamous cell carcinoma: prognostic implications and potential for personalized therapies.

Mitochondrial folate pathway regulates myofibroblast differentiation and silica-induced pulmonary fibrosis.

Pharmacological targeting of one‑carbon metabolism as a novel therapeutic strategy for glioblastoma.

Glioma cells require one-carbon metabolism to survive glutamine starvation.

MYEOV overexpression induced by demethylation of its promoter contributes to pancreatic cancer progression via activation of the folate cycle/c-Myc/mTORC1 pathway.

Comparative profiling of single-cell transcriptome reveals heterogeneity of tumor microenvironment between solid and acinar lung adenocarcinoma.

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